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🫀 Lipid Panel Interpretation

Reference cut-offs for TC/LDL/HDL/TG/non-HDL with LDL targets by ASCVD risk and high-triglyceride management. Browser-side reference.

Clinical takeaway

LDL target follows ASCVD risk stratification, which differs slightly by guideline.

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When to use

Interpret a lipid panel against the patient's risk level, not a single fixed cut-off.

How it works

LDL targets: very high < 1.4, high < 1.8, moderate < 2.6, low < 3.4 mmol/L. Non-HDL target ≈ LDL target + 0.8 mmol/L.

Key points

  • LDL target follows ASCVD risk stratification, which differs slightly by guideline.
  • Severe hypertriglyceridaemia (≥ 5.6 mmol/L) prioritises pancreatitis prevention.
  • Lp(a) is an independent hereditary risk — measure at least once.
  • ApoB reflects atherogenic particle number for residual-risk assessment.

References

Decision support for licensed clinicians only; not a substitute for clinical judgement, diagnosis or local protocols.

Worked calculation

The values below come from this tool's own example placeholders and are computed server-side with the formula shown on this page, so the arithmetic can be checked quickly. It demonstrates how to substitute values only — it is not clinical advice and not a real case.

ViewReference cut-offs

LipidsLipid reference cut-offs (adult, fasting)

  • LDL-COptimal < 2.6 mmol/L (100 mg/dL); ≥ 3.4 (130) high; ≥ 4.9 (190) very high
  • HDL-CLow: men < 1.0, women < 1.3 mmol/L (< 40 / < 50 mg/dL)
  • TGDesirable < 1.7 mmol/L (150); 1.7–2.3 borderline; ≥ 2.3 high
ViewSpecial markers/secondary

LipidsSpecial markers / secondary causes

  • Lp(a)Hereditary, independent risk — measure at least once
  • ApoBReflects atherogenic particle number, better for residual-risk assessment than LDL
  • Non-fastingUsually acceptable; recheck fasting if TG markedly raised or severe hypertriglyceridaemia

Frequently asked questions

What is Lipid Panel Interpretation?
Reference cut-offs for TC/LDL/HDL/TG/non-HDL with LDL targets by ASCVD risk and high-triglyceride management. Browser-side reference.
How is Lipid Panel Interpretation calculated? What is the core formula?
LDL targets: very high < 1.4, high < 1.8, moderate < 2.6, low < 3.4 mmol/L. Non-HDL target ≈ LDL target + 0.8 mmol/L.
When is Lipid Panel Interpretation used?
Interpret a lipid panel against the patient's risk level, not a single fixed cut-off.
What are the key clinical points for Lipid Panel Interpretation?
LDL target follows ASCVD risk stratification, which differs slightly by guideline. Severe hypertriglyceridaemia (≥ 5.6 mmol/L) prioritises pancreatitis prevention. Lp(a) is an independent hereditary risk — measure at least once. ApoB reflects atherogenic particle number for residual-risk assessment.
What are the limits and cautions when using Lipid Panel Interpretation?
For licensed clinicians and clinical researchers. Interpret results with history, investigations and local protocols; not a diagnosis or prescription, and not a substitute for multidisciplinary decision-making or local guidelines.
How is Lipid Panel Interpretation calculated in practice? Can you show a worked example?
Inputs: View Reference cut-offs → Result: Lipids Lipid reference cut-offs (adult, fasting)(LDL-C: Optimal < 2.6 mmol/L (100 mg/dL); ≥ 3.4 (130) high; ≥ 4.9 (190) very high, HDL-C: Low: men < 1.0, women < 1.3 mmol/L (< 40 / < 50 mg/dL), TG: Desirable < 1.7 mmol/L (150); 1.7–2.3 borderline; ≥ 2.3 high) Inputs: View Special markers/secondary → Result: Lipids Special markers / secondary causes(Lp(a): Hereditary, independent risk — measure at least once, ApoB: Reflects atherogenic particle number, better for residual-risk assessment than LDL, Non-fasting: Usually acceptable; recheck fasting if TG markedly raised or severe hypertriglyceridaemia)

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